It is well understood that myopia commonly onsets in early childhood and continues to progress until late teenage years or early adulthood. The primary structural change in myopia is axial elongation beyond the intended eye length. As a result of the longer than normal eye length, the eye is at risk of developing complications such as retinal break and detachment, myopia maculopathy, choroidal neovascularization and glaucoma, especially in later life and the risk increases exponentially with the degree of myopia.[1, 2]

Therefore, a key goal for myopia control is to control or slow progression to reduce the risk of high myopia.

In a myopic eye, there is good correlation between axial length and the degree of myopia. Therefore, both are good indicators in assessing the rate of progression of myopia. It is now well understood that myopia progression is faster in younger children and slows with age.[3] The younger the age of onset of myopia, the greater the risk. In addition, ethnicity, parental myopia, and schooling are factors that were found to influence progression.